Oops, you're using an old version of your browser so some of the features on this page may not be displaying properly.

MINIMAL Requirements: Google Chrome 24+Mozilla Firefox 20+Internet Explorer 11Opera 15–18Apple Safari 7SeaMonkey 2.15-2.23

Poster display session: Biomarkers, Gynaecological cancers, Haematological malignancies, Immunotherapy of cancer, New diagnostic tools, NSCLC - early stage, locally advanced & metastatic, SCLC, Thoracic malignancies, Translational research

2242 - Clinical characteristics of ovarian cancer relapse in BRCA1/2 germ-line mutation carriers and non-carriers

Date

20 Oct 2018

Session

Poster display session: Biomarkers, Gynaecological cancers, Haematological malignancies, Immunotherapy of cancer, New diagnostic tools, NSCLC - early stage, locally advanced & metastatic, SCLC, Thoracic malignancies, Translational research

Topics

Genetic Testing and Counselling;  Targeted Therapy;  Genetic and Genomic Testing

Tumour Site

Ovarian Cancer

Presenters

Anna Sokolenko

Citation

Annals of Oncology (2018) 29 (suppl_8): viii332-viii358. 10.1093/annonc/mdy285

Authors

A.P. Sokolenko, T.V. Gorodnova, A.O. Ivantsov, I.V. Berlev, E.N. Imyanitov

Author affiliations

  • Department Of Tumor Growth Biology, N.N. Petrov Institute of Oncology, 197758 - Saint-Petersburg/RU
More

Abstract 2242

Background

Approximately 15% of ovarian cancer (OC) incidence is attributed to germ-line mutations in BRCA1 or BRCA2 genes. Being a distinct biological subset of OC disease, BRCA1/2-driven cancers are usually characterized by good response to chemotherapy. However, even if OC patient undergoes complete surgical cytoreduction and potentially effective systemic therapy, the probability of the tumor relapse is high. This study aimed to compare some essential clinical features of OC relapses in hereditary vs. sporadic OC.

Methods

We identified 212 women with relapsed high-grade serous OC, who were treated in the N.N. Petrov Institute of Oncology within years 2000-2014, underwent complete primary (n = 113) or interval (n = 99) surgical debulking, and had available clinical data for analysis. 66 women were BRCA1/2 germ-line mutation carriers and 146 were mutation negative. Recurrences were classified according to anatomical location (local, regional, distant, marker), type (systemic or discrete) and pattern of spread.

Results

As expected, median PFI (platinum-free interval) in BRCA1/2 carriers was longer as compared to sporadic cases (13.2 months vs. 8.0 months) [p < 0.001]. The proportion of OC cases with PFI > 12 months was significantly higher among BRCA1/2 carriers [38/66 (58%) vs. 51/146 (35%), p = 0.003]. There was no statistical difference in the frequency of distant relapses between these groups [10/66 (15%) vs. 31/146 (21%), p = 0.4]. Systemic recurrences (i.e., multiple lesions) occurred significantly more frequently in sporadic cases as compared to patients with BRCA1/2 mutation [98/144 (68%) vs. 30/60 (50%), p = 0.02] and were associated with shorter duration of PFI [p = 0.003]. The proportion of patients who could be subjected to the local treatment (locoregional discrete recurrence with lymphatic/transcoelomic spread) was higher among BRCA1/2 mutation carriers than non-carriers [29/66 (44%) vs. 45/146 (31%), p = 0.045].

Conclusions

BRCA1/2-driven OC are characterized by more favorable mode of relapse than sporadic high-grade serous ovarian cancers.

Clinical trial identification

Legal entity responsible for the study

N.N. Petrov Institute of Oncology.

Funding

Russian Science Foundation [grant number 14-25-00111].

Editorial Acknowledgement

Disclosure

All authors have declared no conflicts of interest.

This site uses cookies. Some of these cookies are essential, while others help us improve your experience by providing insights into how the site is being used.

For more detailed information on the cookies we use, please check our Privacy Policy.

Customise settings
  • Necessary cookies enable core functionality. The website cannot function properly without these cookies, and you can only disable them by changing your browser preferences.