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Poster display session: Biomarkers, Gynaecological cancers, Haematological malignancies, Immunotherapy of cancer, New diagnostic tools, NSCLC - early stage, locally advanced & metastatic, SCLC, Thoracic malignancies, Translational research

4204 - Bone metastasis reduces responsiveness to EGFR-TKIs in patients with EGFR-mutated advanced lung adenocarcinoma

Date

20 Oct 2018

Session

Poster display session: Biomarkers, Gynaecological cancers, Haematological malignancies, Immunotherapy of cancer, New diagnostic tools, NSCLC - early stage, locally advanced & metastatic, SCLC, Thoracic malignancies, Translational research

Presenters

Xiaobing Zheng

Citation

Annals of Oncology (2018) 29 (suppl_8): viii493-viii547. 10.1093/annonc/mdy292

Authors

X. Zheng, C. Huang, G. Lin

Author affiliations

  • Department Of Thoracic Oncology, Fujian Cancer Hospital, 350014 - Fuzhou/CN
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Abstract 4204

Background

The impact of bone metastasis on the patient’s response to EGFR-TKI therapy is yet to be determined in lung adenocarcinoma (LUAD) patients harboring EGFR mutations.

Methods

This is a retrospective analysis of the efficacy of EGFR-TKIs for LUAD patients with EGFR mutations, comparing patients with bone metastases (BM) and those with no bone metastases (NBM). Regular imaging that evaluated the bone metastasis response was required for patients with BM. Overall response, median progression-free survival (mPFS), and progression patterns were calculated.

Results

Of the 502 patients reviewed, 175 were evaluated (BM 96, NBM 79). Clinical characteristics were balanced between the groups. Median PFS was 11.0 months in the NBM group versus 7.0 months in the BM group, P = 0.013. The mPFS was significantly decreased in patients with multiple BM (≥4) compared with those having oligometastases (1-3) or NBM (7.0 months vs. 10.0 months vs. 11.0 months, P = 0.003). Multivariate analysis confirmed that bone metastasis was an independent negative predictive factor of PFS, hazard ratio 1.87, P = 0.003. The response rate was higher in the NBM group than in the BM group (70.9% vs. 56.3%), P = 0.046. Bone was one of the frequent sites of EGFR-TKIs failure, accounting for 52 of 125 (41.6%). Patients with NBM had a remarkably lower rate of bone failure compared to patients with BM, 11.8% vs. 62.2%, P < 0.001.

Conclusions

Bone metastasis and multiple metastases in particular, reduce the responsiveness to EGFR-TKIs for LUAD patients with EGFR mutations. Monitoring of bone metastases should be a routine clinical practice in patients with BM.

Clinical trial identification

Legal entity responsible for the study

Fujian Cancer Hospital.

Funding

Fujian Provincial health systemic youth backbone training projects [grant number 2015-ZQN-ZD-9].

Editorial Acknowledgement

Disclosure

All authors have declared no conflicts of interest.

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